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Video and creativity

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The ‘Creativity’ theme of the Viducate Project is one of three inter-related and overlapping concerns which inform our overall work. We decided to emphasise the ‘creative’ theme first because it allowed for exploration of understandings of the term creativity in a range of educational contexts. In order to formalise and discuss our approaches to this central theme, it is first necessary to introduce a brief discussion of what is meant by creativity and why it is important for the project.

This page will give you an overview of how we have outlined the viducate theme of creativity, what examples we have collected and where you can get involved. You can download below:

  • The introduction to the creativity theme
  • The join-in task for the creativity theme
  • The current documentation of the theme

If you are interested in the stop motion idea you can download the "" from Chris Munsey which he has used for the "We to to the movies" project. And also the "", a simple animation exercise where no video technology is needed!

Attachments:
[introduction creativity theme SV]548 Kb
[introduction creativity theme]208 Kb
[introduction creativity theme LT]164 Kb
[introduction creativity theme DE]480 Kb
[introduction creativity theme ES]548 Kb
[introduction creativity theme EL]559 Kb
[introduction creativity theme FR]473 Kb
[join-in activity moving objects DE]778 Kb
[join-in activity moving objects ES]872 Kb
[join-in activity moving objects]774 Kb
[join-in activity moving objects LT]961 Kb
[join-in activity moving objects EL]809 Kb
[join-in activity moving objects FR]752 Kb
[join-in activity moving objects SV]700 Kb
[rabbits flipbook]843 Kb
[thaumatrope - animation exercise]605 Kb
 

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Viducate, Robert Ferguson

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  • ED can in many cases be treated by drugs taken orally, injected, or as penile suppositories. These drugs increase the efficacy of NO, which dilates the blood vessels of corpora cavernosa. When oral drugs or suppositories fail, injections (e.g. of apomorphine) into the erectile tissue of the penile shaft may work. Pulmonary hypertension is caused by high blood pressure in the arteries that supply the lungs with blood. People who suffer from the condition can become tired, dizzy and short of breath, because the arteries feeding the lungs constrict and reduce the supply of oxygenated blood being circulating throughout the body. Sildenafil is the first oral agent to be well documented as an effective form of treatment for men experiencing ED. Since its introduction in March, 1998, no other therapy for ED has achieved such prominent public recognition. Of the 250,000 American physicians who have written prescriptions for sildenafil, 14% have been written by urologists and 82% by nonurologists. The hormonal birth control patch (brand name: Ortho Evra) uses estrogen and progestin to prevent pregnancy. It is applied to 1 of 4 places -- the buttocks, abdomen, upper torso or outer arm. The patch is used on a 4-week cycle. You put on a new patch once a week for 3 weeks. You don't use a patch during the 4th week and your period will start. Although sildenafil can increase cGMP in T cells, DCs, and CD11b+ cells (Fig. 5), the following data indicate that Gr-1+/CD11b+ MDSCs are its primary cellular target. Gr-1 depletion does not augment sildenafil-mediated antitumor activity (Fig. 6 E), and sildenafil down-regulates MDSC suppressive pathways in vivo (Fig. 6, BD). Moreover, sildenafil reverses MDSC suppression in vitro (Fig. 7). MDSCs and/or tumor-associated macrophages have been shown to induce apoptosis or anergy in CD8+ and CD4+ T cells through NOS2- and/or ARG1-dependent mechanisms (34). In fact, NO production anergizes Th1 cells through inhibition of IL-2 signaling (34). Alternatively, in a mixed Th1/Th2 cell environment where ARG-induced pathways also mediate immunosuppression, MDSCs produce NO and super-oxide radicals to generate peroxynitrites that induce apoptosis of activated CD8+ T cells (9). A greater understanding of the role of MDSCs in tumor-induced immune dysfunction (7, 42) will establish the scientific rationale for a targeted pharmacologic approach to disrupt these suppressive mechanisms and may serve as an adjunct to immunotherapy. We previously showed that nitroaspirin could abrogate the inhibitory activity of MDSCs by enhancing the preventive and therapeutic efficacy of antitumor vaccines (43). However, despite its use as a vaccine adjuvant, nitroaspirin demonstrated no antitumor efficacy when used alone. In contrast, down-modulation of both ARG1 and NOS2 in MDSCs (Fig. 6) with PDE5 inhibitors effectively abrogates MDSC-mediated immune suppression, resulting in a measurable antitumor response (Fig. 1, Fig. 3, and Fig. 4). We have recently shown that to effectively exert their suppressive function, MDSCs must (a) be activated by IFN- production from antigen-stimulated T cells, (b) release their own IFN-, and (c) be responsive to IL-13 (29). Cooperation between these two cytokines leads to the activation of ARG1 and NOS2 enzymes. Sildenafil neither alters IFN- production from activated lymphocytes (not depicted) nor changes IL-13 and IFN- production from MDSCs (Fig. S6, available at http://www.jem.org/cgi/content/full/jem.20061104/DC1). Rather, PDE5 inhibition down-regulates IL-4R expression on MDSCs (Fig. 5 and Fig. 6), likely impairing their responsiveness to IL-13. Acknowledgments We thank Sergio Martinez for constructing Fig. 3. buy generic propecia where to buy viagra canada Q. How does a VED device work? Erectile Dysfunction is defined as the inability to achieve or maintain an erection sufficient for mutually satisfying intercourse. Erectile Dysfunction impacts more than a man's sexual activity. The emotions and uncertainties that coincide with this condition often have a significant effect on a man's self-esteem, as well as and his relationship with his partner. Although the incidence of Erectile Dysfunction increases with age, it is not an inevitable result of aging. Erectile Dysfunction is a secondary condition linked to many medical conditions. The new research suggests that sildenafil may help all chronic obstructive pulmonary disease (COPD) patients -- even those not diagnosed with full-blown PAH -- who experience potentially dangerous pulmonary arterial blood pressure increases both at rest and following exercise. Erectile dysfunction. This inflatable penile prosthesis has fluid located at the base of the device. When the tip of the prosthesis is squeezed, the fluid is transferred into the cylinder. Q. Does the VED have any advantages? The primary feat of alcohol is increasing blood volume, which in turn causes higher blood pressure. The state lasts till the unnecessary liquid has been removed from the blood by means of urine. So, in theory, alcohol intake ought to help out erection and not cause erectile dysfunction. "Moreover, men who are physically inactive and had high rates of sedentary behavior, such as watching three or more hours of TV per day, were much more likely to have erectile dysfunction compared with men who were physically active," Selvin said, so, "increasing exercise may be an effective non-pharmacologic treatment." Abstract Erectile dysfunction (ED) is a clinical disorder that results from a continuous spectrum of clinical factors, including physical illness (comprising the organic component of ED), reaction to stress (the intrapsychic component of ED) and relationship difficulties (the relationship component of ED). Testosterone clearly has a relevant role in all three causes of ED; the usefulness of this hormone in the treatment of ED has not, however, been completely clarified. The main physiological action of testosterone in the male sexual response is to regulate the timing of the erectile process as a function of sexual desire, thereby coordinating penile erection with sex. The link between ED, hypogonadism and underlying disorders (such as metabolic syndrome and type 2 diabetes mellitus) is nowadays well documented. The recognition of underlying disorders might be useful in motivating men with ED to improve their health-related lifestyle choices. Hence, patients with ED might be considered 'lucky', because their disorder offers the opportunity to undergo medical examinations to detect underlying disease. Both ED and hypogonadism are treatable conditions. A range of testosterone preparations are available for supplementation; their combination with phosphodiesterase 5 inhibitors might improve outcomes in some cases. PDE5 inhibition restores T cell proliferation in MM and head and neck cancer patients

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