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Viducate is about video in education and the best starting point is our . The gallery contains selected material linked with the themes of creativity, active citizenship and intercultural communication. It also offers video documenting the project and interviews with partners.
The gallery provides also further pedagogical background information - who has produced the video, what is behind it and how can it be transferred to other educational sectors or learning environments.
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If erection problems seem to be caused by a medication you are taking for an unrelated condition, consult your doctor. You may benefit from reducing the dose of the drug or changing to another drug that has the same result but not the same side effects. DO NOT change or stop taking any medications without first talking to your doctor.
The Erectile Dysfunction-Heart Disease Connection
From the Internal Medicine Unit, IRCCS Maugeri Foundation Hospital, Department of Internal Medicine and Medical Therapeutics (C.G., S.G., E.D.A., G.B., P.F.), Cardiology Unit (C.F.), Internal Medicine, Vascular and Metabolic Diseases, IRCCS Policlinico San Matteo (D.G.), and the Department of Internal Medicine and Medical Therapeutics (S.B.S.), University of Pavia; and Diabetes Centers of Pavia and Mede (A.G.), Azienda Ospedaliera Province of Pavia, Pavia, Italy.
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The ability to achieve and sustain erections require:
If you have difficulty having or keeping an erection more than 25% of the time, it is considered a problem.
Apomorphine is a sublingual agent that is not approved by the US Food and Drug Administration (FDA). Apomorphine has a central effect on the hypothalamus. It is a D1/D2 dopamine receptor agonist from the apomorphine (nonopiate) drug class. This agent has been shown to specifically activate c-fos gene expression in the paraventricular and supraoptic nuclei of the hypothalamus in animal models. These areas are known to be involved with penile erections.
An erection begins in the brain. Physical and/or mental stimulation cause nerves in the brain to send chemical messages to nerves in the penis telling the penile blood vessels to relax so that blood can flow freely into the penis. Once in the penis, high pressure traps the blood within both corpora cavernosa to sustain an erection. This causes the penis to expand, creating an erection.
PDE-5 is an enzyme found in trabecular smooth muscle. It catalyzes the degradation of cGMP, which results in an elevated cytosolic calcium concentration and smooth-muscle contraction (Fig. 2). PDE-5 inhibitors, therefore, block this biochemical pathway to promote erection. Eleven subtypes of PDEs have been described, with 30 isoenzymes mediating a variety of physiologic actions throughout the body.32 PDEs exhibit enormous functional diversity; at present, our understanding of PDE types 1 to 6 are considerably better than our understanding of PDE types 7 to 11.
13 Common Sex-Drive Killers
Penile blood flow is controlled by the autonomic erection centre, which provides parasympathetic (S2–S4) and sympathetic (T12–L2) input to the pelvic plexus,8 including the cavernous nerves that innervate the cavernosal arteries and trabecular smooth muscle. These nerves are responsible for the delivery of high local concentrations of nitric oxide to the trabecular smooth muscle, which results in relaxation (Fig. 2).9,10 Nitric oxide diffuses across the smooth-muscle membrane and activates guanylate cyclase to produce cyclic guanosine monophosphate (cGMP); the biochemical cascade that ensues results in altered potassium and calcium ion channel permeability; ultimately, the decrease in cytosolic calcium concentration causes smooth-muscle relaxation and increased regional blood flow.11,12 Phosphodiesterase enzymes (PDEs) regulate this pathway by inactivating cGMP, which results in elevated cytosolic calcium concentrations and smooth-muscle contraction. PDE type 5 is the most important isoenzyme in the corpora cavernosa.13,14,15
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